Abstract
Heart remodeling processes in the development of chronic heart failure (CHF) play a key role in systemic damage to organs and systems. With worsening of CHF, the linear velocities of the liver blood flow (bf) decrease, the diameters of both the common hepatic artery and the portal vein increase. Changes in arterial bf in patients with CHF had a multidirectional character from group to group, narrowing of the lumen of the vessel at the initial stages of the disease and its progressive expansion to functional class IV, fully reflect changes in central hemodynamics, and are explained by the structure of the vessel and its lability in relation to neurohumoral influences characteristic of the development of CHF.